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Upon activation, phosphorylated MLKL (p-MLKL) oligomerizes and translocates to the cell membrane, forming pores that disrupt electrochemical gradients, resulting in lytic cell rupture and the release of damage-associated molecular patterns (DAMPs), inflammatory cytokines and chemokines
1 Introduction Coronary heart disease (CHD) represents a significant threat to global health, characterized by complex and multifactorial pathogenesis ( The pathophysiological significance of ferroptosis in cardiovascular disease stems from its multifaceted involvement in abnormal iron metabolism, lipid peroxidation, and ROS accumulationprocesses fundamental to the development of coronary artery disease (CAD)
The solution may look normal but deliver reduced potency
Still, factors like stress, pollution, alcohol, and age can diminish glutathione levels, which ultimately help protect our cells from these factors
